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These effects were reversed by using AMPK siRNAs, confirming the inhibitory function of activated AMPK in 3T3-L1 adipocytes

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Conclusion Although oral B12 therapy evoked an important metabolic response, this response did not persist for an additional three months following cessation of therapy, causing us to question whether extending oral vitamin B12 treatment beyond one month would have a significant effect on the clinical manifestations of cobalamin deficiency